HCV & CTLs
The viral clearance in interferon-treated chronic hepatitis C is associated with
increased cytotoxic T cell frequencies.
J Hepatol 1999 Sep;31(3):407-15
Lohr HF, Schmitz D, Arenz M, Weyer S, Gerken G, Meyer zum Buschenfelde KH
Department of Internal Medicine, Johannes Gutenberg-University Mainz, Germany. [Medline
record in process]
BACKGROUND/AIMS: Cytotoxic T lymphocytes have been demonstrated in
peripheral blood and liver tissue of patients with chronic hepatitis C virus infection,
but their significance for viral clearance is unknown. Therefore, we analyzed hepatitis C
virus-specific cytotoxic T lymphocyte precursor frequencies in chronic hepatitis C virus
carriers during interferon-alpha treatment.
METHODS: Blood mononuclear cells or CD8+ T cells from HLA-A2 positive and
negative patients and controls were analyzed in chromium-release assays using a panel of
18 synthetic peptides from the HCV core, E1 and NS4 antigens bearing HLA-A2 binding
motifs. Specific cytotoxic T lymphocyte precursor frequencies were studied within CD8+ T
cells derived from interferon-alpha-treated patients using a TNF-alpha-based ELISPOT assay
and compared to viremia levels.
RESULTS: T cells from 16 of 24 HLA-A2+ but none of the six HLA-A2-
patients with chronic hepatitis C and six HLA-A2+ healthy controls lysed targets pulsed
with peptide cocktails. Fine specificity revealed four very immunogenic epitopes in the
core (C36-44, C132-140) and the envelope regions (E332-340, E363-372). Cytotoxic T
lymphocyte precursor frequencies were prospectively analyzed in 11
interferon-alpha-treated HLA-A2+ hepatitis C virus patients. Four sustained
and two transient therapy responders showed lower pretreatment viremia levels and
significantly higher specific cytotoxic T lymphocyte precursor frequencies during viral
clearance compared to five therapy non-responders and untreated controls.
CONCLUSIONS: The quantitative induction of HLA-class I restricted
responses by interferon-alpha could contribute to a beneficial outcome of hepatitis C
virus infections. Furthermore, it appears that the balance between viral load and specific
cellular immune responses is critical for successful viral clearance.