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Using Imaging to Understand the Role of Epicardial Adipose Tissue in HIV-Related Atherosclerosis
  Allison G. Hays, M.D.
Assistant Professor of Medicine
Division of Cardiology
Johns Hopkins Hospital
Baltimore, MD, US
18th International Workshop on Co-morbidities and Adverse Drug Reactions in HIV 12–13 September 2016, New York, USA
Reported by Jules Levin



The magnitude of this effect was such that an approximate 8% increase in EAT (epicardial fat) was associated with a 5–6% increase in prevalence of noncalcified plaque. Notably, the relationship fell out after adjustment for visceral fat in the HIV group but not the non-HIV group.
First, they strongly suggest that HIV-infected men have a unique pattern of plaque on CTA, characterized by increases in noncalcified plaque. Second, they demonstrate increased EAT in HIV-infected men, which may relate to increased plaque as well as critical metabolic parameters. As the study by Brener et al. is cross-sectional, we do not know the sequence of development of EAT







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