HCV & HIV Specific CD4 Response
Two Articles:
1. EPIMMUNE RECEIVES GRANTS FROM THE NIH TO FUND HEPATITIS C AND AIDS VACCINE;
press release
2. Recurrence of Hepatitis C Virus After Loss of Virus-Specific CD4(+) T-Cell Response in
Acute Hepatitis C
Gastroenterology 1999 Oct;117(4):933-941
Gerlach JT, Diepolder HM, Jung MC, Gruener NH, Schraut WW, Zachoval R, Hoffmann R,
Schirren CA, Santantonio T, Pape GR Institute for Immunology, Klinikum Groshadern,
University of Munich, Germany.
[Record supplied by publisher]
Background & Aims: The prospective comparison of patients with acute hepatitis C virus
(HCV) who spontaneously clear the virus with those who cannot achieve viral elimination
and progress to chronic hepatitis offers the unique opportunity to analyze natural
mechanisms of viral elimination. Methods: We studied the HCV-specific CD4(+) T-cell
response in 38 patients with acute HCV and correlated the clinical course with the
antiviral immune response. The individual HCV-specific T-cell response was assessed in a
proliferation assay ((3)H-thymidine uptake) and an enzyme-linked immunospot assay.
Results: Patients were classified according to their clinical course and pattern of CD4(+)
T-cell responses in 3 categories: first, patients mounting a strong and sustained
antiviral CD4(+)/Th1(+) T-cell response who cleared the virus (HCV RNA-negative; n = 20);
second, patients who were unable to mount an HCV-specific CD4(+) T-cell response and
developed chronic disease (n = 12); and third, patients who initially displayed a strong
CD4(+) T-cell response and eliminated the virus (HCV PCR-negative) but subsequently lost
this specific T-cell response (n = 6). The loss of the HCV-specific CD4(+) T-cell response
was promptly followed by HCV recurrence. Conclusions: The results indicate that a
virus-specific CD4(+)/Th1(+) T-cell response that eliminates the virus during the acute
phase of disease has to be maintained permanently to achieve long-term control of the
virus. The induction and/or maintenance of virus-specific CD4(+) T cells could represent a
promising therapeutic approach in HCV infection.
EPIMMUNE RECEIVES GRANTS FROM THE NIH TO FUND HEPATITIS C AND AIDS VACCINE
RESEARCH
SAN DIEGO, CA, October 26, 1998 -- Epimmune Inc., a majority-owned subsidiary of Cytel
Corporation (NASDAQ: CYTL), today announced that it has received two Phase II Small
Business Innovation Research (SBIR) grants totaling $1.5 million from the National
Institutes of Health (NIH) to investigate a new approach for treating and preventing viral
infections. The two studies are aimed at extending into non-human primate models the
preclinical evidence that Epimmunes technology enables the induction of specific
immune responses critical to controlling hepatitis C virus (HCV) and human
immunodeficiency virus (HIV) infections.
In spite of recent breakthroughs in anti-viral therapy, developing cures for HCV, HIV and
other viral infections remain unrealized goals. Viruses are extremely resilient.
They use a variety of strategies to divert the immune response and to persist through
months of direct attack by anti-viral drugs, said Robert W. Chesnut, Ph.D.,
Executive Vice President, Research and Development of Epimmune.
Since the human immune system is capable of preventing these viruses from becoming
established, as well as clearing them once established, we have focused on understanding
and mimicking these beneficial immune mechanisms, added Dr. Chesnut. In
particular, CD8+ killer T-cells (CTL) and CD4+ helper T-cells (HTL) have been shown to be
the critical components of the bodys natural defense against infectious diseases
including HCV and HIV. Based on extensive studies of the key receptors that control these
immune responses, we think we have figured out how to design smart vaccines
that will redirect the immune response in such a way that it will effectively attack the
virus and eliminate the infection.
To this end, Epimmune scientists have identified specific viral fragments, known as
epitopes, from the hepatitis C and AIDS viruses, capable of stimulating CTL and HTL.
Epimmunes candidate vaccines are comprised of multiple epitopes from the targeted
virus, selected to focus the immune system on killing the virus while avoiding
non-productive or even counter-productive immune responses.
The NIH grant directed toward HCV vaccine research is a two-year program entitled
Vaccine Approaches to Treatment of Hepatitis C Infection (HCV). Under the
grant, Epimmune will evaluate the feasibility of using chimpanzees to test its
epitope-based vaccine candidates. After validating the chimp as an appropriate model,
Epimmune plans to test the ability of its HCV genetic vaccines to elicit a cellular immune
response targeted to multiple HCV epitopes and determine whether this response can protect
against, or alter the course of, an HCV infection in primates.
The NIH grant directed toward AIDS vaccine research is a two-year program entitled
Peptide-Based Vaccine for Primate Model of AIDS, to be conducted in
collaboration with David I. Watkins, Ph.D., Associate Professor of the Wisconsin Regional
Primate Research Center. Under the grant, the investigators will evaluate the ability of
epitope-based genetic vaccines to elicit a cellular immune response in macaques monkeys
targeted to multiple epitopes from simian immunodeficiency virus (SIV), an AIDS-like
virus, and determine whether this response can protect against, or alter the course of,
the viral infection. Epimmune Inc., established in October 1997, is a majority-owned
subsidiary of Cytel Corporation (NASDAQ: CYTL), operating under separate management and
financing. Applying its substantial immunology expertise and scientific leadership in the
field of T-cell recognition and activation, Epimmune is developing novel vaccines which
stimulate the bodys immune system to treat and prevent infectious diseases and
cancer. Epimmune is collaborating with G.D. Searle & Co., a wholly-owned subsidiary of
Monsanto Co., to develop immune stimulating products for the treatment of cancer.
Additional product targets include prophylactic vaccines for hepatitis C, HIV and malaria
and therapeutic vaccines for hepatitis B, hepatitis C and HIV.